Filtros: Autor es Moreno, V [Restablecer todos los filtros]
Accounting for error due to misclassification of exposures in case-control studies of gene-environment interaction. Statistics in medicine. 2008;27(15):2756-83.
Semiparametric Bayesian modeling of random genetic effects in family-based association studies. Statistics in medicine. 2009;28(1):113-39.
Concentrations and correlations of disinfection by-products in municipal drinking water from an exposure assessment perspective. Environmental research. 2012;114:1-11.
MRE11 deficiency increases sensitivity to poly(ADP-ribose) polymerase inhibition in microsatellite unstable colorectal cancers. Cancer research. 2011;71(7):2632-42.
Genomic and transcriptomic prognostic factors in R0 Dukes B and C colorectal cancer patients. International journal of oncology. 2007;30(5):1099-107.
CLEAR-test: combining inference for differential expression and variability in microarray data analysis. Journal of biomedical informatics. 2008;41(1):33-45.
RiskDiff: a web tool for the analysis of the difference due to risk and demographic factors for incidence or mortality data. BMC public health. 2009;9:473.
COGENT (COlorectal cancer GENeTics): an international consortium to study the role of polymorphic variation on the risk of colorectal cancer. British journal of cancer. 2010;102(2):447-54.
A large-scale meta-analysis to refine colorectal cancer risk estimates associated with MUTYH variants. British journal of cancer. 2010;103(12):1875-84.
Genome-wide association scan identifies a colorectal cancer susceptibility locus on 11q23 and replicates risk loci at 8q24 and 18q21. Nature genetics. 2008;40(5):631-7.
Mutational load distribution analysis yields metrics reflecting genetic instability during pancreatic carcinogenesis. Proceedings of the National Academy of Sciences of the United States of America. 2008;105(11):4306-11.
Common variation in ISL1 confers genetic susceptibility for human congenital heart disease. PloS one. 2010;5(5):e10855.
Genetic and genomic analysis modeling of germline c-MYC overexpression and cancer susceptibility. BMC genomics. 2008;9:12.
Biological convergence of cancer signatures. PloS one. 2009;4(2):e4544.
No association between germline allele-specific expression of TGFBR1 and colorectal cancer risk in Caucasian and Ashkenazi populations. British journal of cancer. 2011;104(4):735-40.
Evidence of linkage to chromosomes 10p15.3-p15.1, 14q24.3-q31.1 and 9q33.3-q34.3 in non-syndromic colorectal cancer families. European journal of human genetics : EJHG. 2012;20(1):91-6.
Tools for protein-protein interaction network analysis in cancer research. Clinical & translational oncology : official publication of the Federation of Spanish Oncology Societies and of the National Cancer Institute of Mexico. 2012;14(1):3-14.
Gene expression differences between colon and rectum tumors. Clinical cancer research : an official journal of the American Association for Cancer Research. 2011;17(23):7303-12.
Clinical Value of Prognosis Gene Expression Signatures in Colorectal Cancer: A Systematic Review. PloS one. 2012;7(11):e48877.
Expression of endoplasmic reticulum stress proteins is a candidate marker of brain metastasis in both ErbB-2+ and ErbB-2- primary breast tumors. The American journal of pathology. 2011;179(2):564-79.
Clinicopathological risk factors of Stage II colon cancer: results of a prospective study. Colorectal disease : the official journal of the Association of Coloproctology of Great Britain and Ireland. 2013;15(4):414-22.
Gene Expression Signature to Improve Prognosis Prediction of Stage II and III Colorectal Cancer. Journal of clinical oncology : official journal of the American Society of Clinical Oncology. 2011;29(1):17-24.
Specific secondary genetic alterations in mantle cell lymphoma provide prognostic information independent of the gene expression-based proliferation signature. Journal of clinical oncology : official journal of the American Society of Clinical Oncology. 2007;25(10):1216-22.